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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1993 1
1994 1
1995 3
1996 3
1997 2
1998 2
1999 2
2000 3
2001 3
2002 2
2003 2
2004 2
2017 4
2021 1
2022 1
2023 1
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32 results

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Page 1
The extracellular matrix protein agrin promotes heart regeneration in mice.
Bassat E, Mutlak YE, Genzelinakh A, Shadrin IY, Baruch Umansky K, Yifa O, Kain D, Rajchman D, Leach J, Riabov Bassat D, Udi Y, Sarig R, Sagi I, Martin JF, Bursac N, Cohen S, Tzahor E. Bassat E, et al. Nature. 2017 Jul 13;547(7662):179-184. doi: 10.1038/nature22978. Epub 2017 Jun 5. Nature. 2017. PMID: 28581497 Free PMC article.
The adult mammalian heart is non-regenerative owing to the post-mitotic nature of cardiomyocytes. The neonatal mouse heart can regenerate, but only during the first week of life. Here we show that changes in the composition of the extracellular matrix during this we …
The adult mammalian heart is non-regenerative owing to the post-mitotic nature of cardiomyocytes. The neonatal mouse heart can …
Exploring digenic inheritance in arrhythmogenic cardiomyopathy.
König E, Volpato CB, Motta BM, Blankenburg H, Picard A, Pramstaller P, Casella M, Rauhe W, Pompilio G, Meraviglia V, Domingues FS, Sommariva E, Rossini A. König E, et al. BMC Med Genet. 2017 Dec 8;18(1):145. doi: 10.1186/s12881-017-0503-7. BMC Med Genet. 2017. PMID: 29221435 Free PMC article.
BACKGROUND: Arrhythmogenic cardiomyopathy (ACM) is an inherited genetic disorder, characterized by the substitution of heart muscle with fibro-fatty tissue and severe ventricular arrhythmias, often leading to heart failure and sudden cardiac death. ...RESULTS: We id …
BACKGROUND: Arrhythmogenic cardiomyopathy (ACM) is an inherited genetic disorder, characterized by the substitution of heart muscle w …
Role of dystrophin isoforms and associated proteins in muscular dystrophy (review).
Culligan KG, Mackey AJ, Finn DM, Maguire PB, Ohlendieck K. Culligan KG, et al. Int J Mol Med. 1998 Dec;2(6):639-48. doi: 10.3892/ijmm.2.6.639. Int J Mol Med. 1998. PMID: 9850730 Review.
Although the most frequent of these disorders, Duchenne muscular dystrophy, is mainly recognized as a disease of skeletal muscle fibers, pathophysiological changes also involve the heart and diaphragm, as well as the peripheral and central nervous system. Thus current rese …
Although the most frequent of these disorders, Duchenne muscular dystrophy, is mainly recognized as a disease of skeletal muscle fibers, pat …
Disruption of heart sarcoglycan complex and severe cardiomyopathy caused by beta sarcoglycan mutations.
Barresi R, Di Blasi C, Negri T, Brugnoni R, Vitali A, Felisari G, Salandi A, Daniel S, Cornelio F, Morandi L, Mora M. Barresi R, et al. J Med Genet. 2000 Feb;37(2):102-7. doi: 10.1136/jmg.37.2.102. J Med Genet. 2000. PMID: 10662809 Free PMC article.
The second mutation in patient 1, inferred because the unaffected father carries the 8 bp duplication, was not found. In patient 1, both heart and skeletal muscle were analysed and showed reduction of all sarcoglycans in both tissues and incorrect localisation of alpha and …
The second mutation in patient 1, inferred because the unaffected father carries the 8 bp duplication, was not found. In patient 1, both …
Dystrophin-glycoprotein complex sequesters Yap to inhibit cardiomyocyte proliferation.
Morikawa Y, Heallen T, Leach J, Xiao Y, Martin JF. Morikawa Y, et al. Nature. 2017 Jul 13;547(7662):227-231. doi: 10.1038/nature22979. Epub 2017 Jun 5. Nature. 2017. PMID: 28581498 Free PMC article.
The Hippo pathway, a conserved kinase cascade, inhibits cardiomyocyte proliferation in the developing heart to control heart size and prevents regeneration in the adult heart. ...In the hearts of mature Mdx mice (which have a point mutation in Dmd)-a m …
The Hippo pathway, a conserved kinase cascade, inhibits cardiomyocyte proliferation in the developing heart to control heart s …
mRNA expression and cDNA sequences of beta- and gamma-sarcoglycans are normal in cardiomyopathic hamster heart.
Hanada H, Yoshida T, Pan Y, Iwata Y, Nishimura M, Shigekawa M. Hanada H, et al. Biol Pharm Bull. 1997 Feb;20(2):134-7. doi: 10.1248/bpb.20.134. Biol Pharm Bull. 1997. PMID: 9057973
In BIO14.6 cardiomyopathic hamster heart, the dystrophin-glycoprotein complex is disrupted and sarcoglycans are greatly reduced in abundance. ...
In BIO14.6 cardiomyopathic hamster heart, the dystrophin-glycoprotein complex is disrupted and sarcoglycans are greatly reduced in ab …
32 results